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Due to their reactivity, high concentrations of ROS can cause oxidative stress by disrupting the balance of antioxidant and prooxidant levels (Zuo et al., 2015)

Modest increases in TRP and KYN have also been observed in the putamen, hippocampus, and temporal cortex, without significant changes in 5-HT or 5-HIAA, indicating that serotonergic metabolism is largely preserved in these regions [236]

Karapetian, R.N., et al., Nuclear oncoprotein prothymosin alpha is a partner of Keap1: implications for expression of oxidative stress-protecting genes

Liposomal glutathione is often praised for its superior absorption, as the glutathione molecule is encapsulated in a lipid layer that protects it through the digestive tract